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Atazanavir
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Atazanavir

Atazanavir is an antiretroviral drug from the class known as protease inhibitors. Protease inhibitors block the activity of the HIV protease (or proteinase) enzyme that HIV uses to break up large viral proteins so that new HIV particles can be formed. Inhibiting this action slows HIV replication and delays damage to the immune system. Atazanavir was developed by Bristol-Myers Squibb, the makers of stavudine (d4T, Zerit) and didanosine (ddI, Videx/VidexEC). It was formerly identified as BM-232632. In the European Union, Atazanavir was approved for use in treatment-experienced people in 2004, at a dose of 300mg, boosted with 100mg ritonavir (Norvir) once a day. It was approved for use in treatment-naive people at the same dose in 2008. In the United States, Atazanavir was approved as an HIV treatment in 2003 without restrictions on its use. The licensed dosing is 400mg once daily for treatment-naive people and 300mg plus 100 mg ritonavir once daily for treatment-experienced people. It is recommended that this drug be taken with food. It has also been approved for use in children six years of age and older.

Other names for this medication:
Atazanavir, Atazor, Reyataz,

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Atazanavir is used together with other medicines for the treatment of the infection caused by the human immunodeficiency virus (HIV). HIV is the virus that causes acquired immune deficiency syndrome (AIDS).

Atazanavir will not cure or prevent HIV infection or AIDS. It helps keep HIV from reproducing and appears to slow down the destruction of the immune system. This may help delay problems that are usually related to AIDS or HIV disease from occurring. Atazanavir will not keep you from spreading HIV to other people. People who receive this medicine may continue to have other problems usually related to AIDS or HIV disease.

This medicine is available only with your doctor's prescription.

This product is available in the following dosage forms:

  • Capsule
  • Powder

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A head-injured patient: caloric needs, clinical progress and nursing care priorities. Conversion of poly(ADP-ribose) polymerase activity to NAD-glycohydrolase during retinoic acid-induced differentiation of HL60 cells. ANC estimates understate female HIV prevalence in this low fertility population but, here, the primary cause is not selection of pregnant women. Evolutionary fate of an unstable human minisatellite deduced from sperm-mutation spectra of individual alleles.

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The temporal relationship between the luteinizing hormone surge and human oocyte maturation. Further studies on the efficacy of an inactivated African horse sickness serotype 4 vaccine. Furthermore, a questionable change of short-latency SEPs could often be decidedly abnormal by finding extreme asymmetry of long-latency components. An MR angiography scan was performed, in which the homolateral middle cerebral artery and internal carotid artery were not identified.

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Does cognitive function influence alaryngeal speech rehabilitation? The pointing pattern was not uniform across the visual field in some of the strabismics, but was composed of areas of larger and smaller pointing errors. Physiological and behavioral responses of New Zealand hypertensive and normotensive rats to stress.

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Diabetes is the extreme manifestation of a spectrum conditions in which the balance of insulin secretion and insulin action (or insulin resistance) has been altered. These results, together with data from other studies on PDE3B and lipolysis in diabetic humans and animals, suggest a novel pathway by which ceramide induces insulin resistance. This in silico 3D-FEA shows that Ti-15Mo plates have greater load-bearing capability. Here, we review the available evidence about the P479L variant and apply to these data the epidemiological criteria for assessing causal associations. Arthroscopy of the PIP and MP joints is useful in assessing articular cartilage and synovium and for synovial biopsy.

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